Can Dental Implants Trigger Autoimmune Disease?

Some research suggests that metal medical and dental implants may be capable of causing an autoimmune reaction in people with metal allergies and other genetic predispositions. A classic model for the induction of chemical sensitivity has been developed, which suggests that the surgical use of artificial implants can induce autoimmune diseases and chemical sensitivity. Animal and human studies conducted 30 years ago on artificial organs showed tissue reactions that caused local fibrosis and the activation of coagulation mechanisms. This was particularly evident when metal, Teflon, and Dacron were first used for heart valve replacement. To prevent this potentially life-threatening condition, anticoagulation is still being used.

Initially, the use of artificial implants was limited to procedures that endangered the person's life, in which the individual died or lost an important organ. Therefore, this fibrotic or coagulant complication and long-term anticoagulant treatment were justified. As surgery became more expansive, it was thought that silicone injections and then silicone implants could be used for cosmetic purposes. However, silicone injections in the face and breasts often lead to many complications, such as severe inflammation and tissue shedding. After these silicone implants, synthetic meshes made of various plastic materials were used to repair large hernias.

These synthetic meshes were moderately successful, and their use was extended to repair smaller openings. It is essential that each patient recognize the range of symptoms that may be related to the presence of a medical or dental metal implant or device in their body, such as the development of an autoimmune disease. This is because an unsafe removal process for metal implants can cause serious injury (and even death) to the patient, in addition to increasing exposure to metal. If this is not recognized, harmful consequences for the local and systemic health of the host can occur, which are hidden by the apparent success of a “stable” implant. To discern the cytokine patterns found in the jaws of patients in the corresponding author's dental office, 14 patients who were diagnosed with FDOJ at the T-IMP sites underwent surgery in the affected area of the mandible, including the removal of existing T-IMPs. In a healthy control group (n %3D 1), healthy jaw samples (HJB) were extracted in the form of perforation cores during routine dental implantation surgery. Metal implant syndrome is particularly seen in jaw, hip and knee implants, as well as in screws, plates, and other metals that are often used in patients with injuries.

In addition to the particles released by implant wear and fatigue, “titanium sensitization” is also the result of the increase in the pro-inflammatory reactivity of nonspecific immune cells (tissue macrophages, monocytes), which in some patients occurs after contact with particulate waste. For example, at the Dallas Environmental Center, more than 200 patients who developed documented food and chemical sensitivities after receiving breast implants have been treated. In other people with chronic diseases, it is necessary to perform skin tests on all the components of the implant using a provocative neutralization technique, since some of the minor metals can cause severe pain, such as molybdenum. Although titanium (TI) is considered to be a compatible implant material, there is growing concern that dissolved titanium particles will induce inflammatory reactions around the implant. Several researchers have discovered that titanium implants can induce inflammation in surrounding tissue over time, leading to the expression of certain mediators known to cause local and systemic health problems. The patients' macrophages were exposed to TI alloy particles extracted from the connective tissue surrounding the hip implants.

Noah Williams
Noah Williams

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